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Archived Comments for: The M50I polymorphic substitution in association with the R263K mutation in HIV-1 subtype B integrase increases drug resistance but does not restore viral replicative fitness

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  1. Acknowledgement of research support from Merck, Inc.

    Mark Wainberg, Lady Davis Institute, Jewish General Hospital

    1 April 2014

    This work should have also cited research support provided by Merck, Inc. We regret this omission.

    Mark Wainberg

    Competing interests

    I have no competing interests.